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The
Continuous Cycle
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How
Anabolics Work:
Hear is the last bit of "bio-chemistry" information we
will discuss before immersing ourselves in the ultra-powerful ON
CYCLE and OFF CYCLE stack! In order to better understand how they
work together, we will give you a little more background information
about how anabolics actually work within the body. As mentioned earlier,
cell receptor sites, including steroid and other growth signaling
receptors, react with anabolic agents in very complex ways. First
off, in order for an anabolic agent to arrive effectively at a receptor
site, it must be ingested and carried by the blood to the desired
location. Any enhancement in the delivery vehicle or transport mechanism
is desired to achieve this optimally. Upon reaching the receptor
site, the cell either lets the anabolic agent pass by or it will
allow the anabolic molecule to latch on to it and potentially exert
its powerful effects on the cell. The targeted cells of interest
for anabolics, if interested in building more muscle of course, would
be skeletal muscle cells. To best visualize this process, think of
receptor sites as having different shapes such as circles, squares,
triangles etc... Now, think of an anabolic agent as having the specific
shape that fits into the receptor shape, which will then enable activation
of the site. Once these shapes match up and bind to the receptor
site, anabolics are believed to form a special bond with the muscle
cell receptor called a cell-signaling complex. This enables it to
send the muscle-building message to the cell nucleus.
Now, once this steroid receptor complex is formed, it travels to
the center of the cell called the nucleus. It then binds with DNA
(deoxyribonucleic acid) and the process of gene expression and cell
transcription are multiplied. The next step of the process is the
duplication of the cells DNA in the form of mRNA (ribonucleic acid),
which is able to leave the nucleus and enter the cytoplasm of the
cell where transcription of messenger mRNA takes place. This then
allows for more protein synthesis to occur, which is initiated by
the signaling of more growth of muscle cell myofibril bands and cell
proliferation due to training stressors. |
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